| Author | Message | |||
     Pedalpro Advanced Member Username: Pedalpro Post Number: 25 Registered: 07-2008 |
I have "grown" out of my trusty spirotiger sport. Need the capability to go larger than 3.5 liter bag so I am going buy the new model. Includes: 2, 2.5, and 3 liter bags. Case. Manuals. Mouthpiece. Nose clip. Face mask. Pulse Oxymeter. $600 Thanks for shopping! | |||
| Danz Senior Member Username: Danz Post Number: 58 Registered: 08-2006 |
Pedalpro...can you e-mail if it's still available... danzucconi@hotmail.com Thanks | |||
| Peterg Senior Member Username: Peterg Post Number: 33 Registered: 03-2008 |
I have my old Spiro Still great shape. Mouthpeice barely used. 3 bags Blue Case ... Asking $650 CAN but will entertain offers !!!! | |||
| Pedalpro Senior Member Username: Pedalpro Post Number: 27 Registered: 07-2008 |
Mine is still available too if Peter has too many buyers. Just in time for relieving Holiday stress. Breathing is good! | |||
| Csuter New member Username: Csuter Post Number: 1 Registered: 10-2010 |
Hi , pedalpro, I am interested in buying your used spirotiger please email me christine@c2skymultisport.com | |||
| Juerg Senior Member Username: Juerg Post Number: 2942 Registered: 04-2006 |
Here a short help for all new users of the above Spiro Tiger.
Now one of the discussion you will encounter is unfortunately still very alive. Here from an otherwise incredible great website with incredible great articles. : Is ventilation volume a limiting factor to maximal endurance? Sometimes you hear people say "I ran out of wind." Is that really possible? Can we reach a point in exercise when ventilation just can't keep up with demand? The answer is no, assuming you don't have acute asthma or some other severe pulmonary dysfunction. We can measure a person's maximal voluntary ventilation (MVV), the maximal volume of air they can breath in and out while at rest, and compare it with their maximal ventilation during exercise. What we see is that untrained people only use about 60 to 85% of their maximum ventilatory capacity even at maximal exercise. For example the MVV for an average male might be nearly 200 l/min. However, during a treadmill VO2 max test, they reach a peak ventilation of only 140 l/min. Highly trained athletes use more of their capacity, perhaps over 90%, but ventilation capacity is still not a limitation on performance. Unlike the story with cardiac output, even during maximal exercise, the ventilatory capacity is not maxed out. " MVV is the amount of air you move per minute. Now if you make a 400 m all out running test you may have perhaps around 60 seconds ( 1 min ) now you run a marathon and you will never even be close to the speed you can run in the 1 min run test. Conclusion is true , speed is never a limitation in a marathon. So in comparison you make a 1 min all out respiration test so yes your MVV is clearly much bigger thna you ever will have in an endurance run or bike. Does that mean your respiration is never the limitation ?. So you run 1 min = 400 m you really never have to train endurance for a marathon , as you can run much faster thna your highest speed in a marathon ???? So you never need to train your breathing as in 1 min you can breath far more as you ever can see in an all out marathon ???? You make up your own thoughts on that commonly used and still used idea on respiration in our university educational systems. ??? | |||
| Juerg Senior Member Username: Juerg Post Number: 2943 Registered: 04-2006 |
Here is a very small case study we did last week to see the "team work" and what it means in the overall picture instead of testing just MVV. Look at the connection of VE as a classical ( "ventilatory reaction " and the reaction in TSI % at the same point. We may have to go back to the grass root of exercise physiology and look at the whole team and not in the microbiological level ( this is important to but of little use in the big picture of coaching ) here to enjoy .   So there are many reasons why respiration can be limited far before you ever reach a tested MVV. It can already have an influence by simply changing position. Here a very very basic study to show that . " PULMONARY PERFUSION IN THE PRONE AND SUPINE POSTURES IN THE NORMAL HUMAN LUNG G. Kim Prisk,1,2 Kei Yamada,3 A. Cortney Henderson,1 Tatsuya J. Arai,1 David L. Levin,2* Richard B. Buxton,2 and Susan R. Hopkins1,2 1 Department of Medicine, University of California, San Diego, La Jolla CA, 92093 2 Department of Radiology, University of California, San Diego, La Jolla CA, 92093 3 School of Medicine, University of California, San Diego, La Jolla CA, 92093 Address for Correspondence: G. Kim Prisk Ph.D., D.Sc., Department of Medicine, 0931, University of California, San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0931, Phone 858-455-4756, FAX 858-455-4765,Email: kprisk/at/ucsd.edu *Current address for D.L. Levin: Department of Radiology, Mayo Clinic, Rochester, MN 55905 The publisher's final edited version of this article is available free at J Appl Physiol. References AbstractProne posture increases cardiac output and improves pulmonary gas exchange. We hypothesized that in the supine posture, greater compression of dependent lung limits regional blood flow. To test this, MRI-based measures of regional lung density, MRI arterial spin labeling quantification of pulmonary perfusion, and density-normalized perfusion were made in 6 healthy subjects. Measurements were made in both the prone and supine posture at FRC. Data were acquired in 3 non-overlapping 15mm sagittal slices covering most of the right lung: central, middle and lateral, which were further divided into vertical zones: anterior, intermediate and posterior. The density of the entire lung was not different between prone and supine, but the increase in lung density in the anterior lung with prone posture was less than the decrease in the posterior lung (change: +0.07 g/cm3 anterior, −0.11 posterior; P<0.0001) indicating greater compression of dependent lung in supine posture, principally in the central lung slice (P<0.0001). Overall density-normalized perfusion was significantly greater in prone posture (7.9±3.6 ml/min/g prone, 5.1±1.8 supine, a 55% increase, P<0.05) and showed the largest increase in the posterior lung as it became non-dependent (change: +71% posterior, +58% intermediate, +31% anterior, P=0.08), most marked in the central lung slice (P<0.05). These data indicate that central posterior portions of the lung are more compressed in the supine posture, likely by the heart and adjacent structures, than are central anterior portions in the prone, and that this limits regional perfusion in the supine posture." |
